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|Peptide Content:||Ý80.0%||Water Content:||¡Ü8.0%|
|TrifluoroAcetate Content:||¡Ü12.0%||Molecular Formula:||C98H138N24O33|
Supply High Purity Bivalirudin Tfa Bivalirudin Trifluoroacetate CAS 128270-60-0 White Powder
Please contact: Yvonne Bai
Whatsapp: +86 18872220653
|Purity (HPLC):||98% Min|
|Appearance :||White powder|
Bivalirudin is a DTI that overcomes many limitations seen with indirect thrombin inhibitors, such as heparin. Bivalirudin is a short, synthetic peptide that is potent, highly specific, and a reversible inhibitor of thrombin.It inhibits both circulating and clot-bound thrombin,while also inhibiting thrombin-mediated platelet activation and aggregation. Bivalirudin has a quick onset of action and a short half-life.It does not bind to plasma proteins (other than thrombin) or to red blood cells. Therefore it has a predictable antithrombotic response. There is no risk for Heparin Induced Thrombocytopenia/Heparin Induced Thrombosis-Thrombocytopenia Syndrome (HIT/HITTS).It does not require a binding cofactor such as antithrombin and does not activate platelets.These characteristics make bivalirudin an ideal alternative to heparin.
Product Name :Bivalirudin Trifluoroacetate
CAS number :128270-60-0
Place of origin :Shanghai Shenzhenor Guangzhou
Grade standard:Medicine grade
Form :White powder
Bivalirudin Trifluoroacetate Description:
Bivalirudin is a specific and reversible bivalent direct thrombin inhibitor. Bivalirudin specifically binds to both the catalytic site and to the anion-binding exosite of circulating and clot-bound thrombin.
Bivalirudin is a synthetic 20 residue peptide (thrombin inhibitor) which reversibly inhibits thrombin. in vitro: Eptifibatide (8 mg/mL) added together with a low (70 ng/mL) concentration of bivalirudin (a direct thrombin inhibitor) effectively (approximately 90%) reduced platelet aggregation induced by thrombin (0.2 U/mL) In thrombin generation assay (TGA), bivalirudin had no effect on these parameters up to 10 μmol/l Bivalirudin-facilitated binding of MPO to BAEC resulted also in functional changes in terms of increased NO consumption as well as enhanced MPO-mediated redox modifications in vivo: The use of bivalirudinprevented further increase in antiheparin/PF4 antibody IgG levels in rats Three animals in the 500-mg/kg/24 h group, and 7 animals in the 2000-mg/kg/24 h group in the toxicokinetic assessment phase of the study were found dead or euthanized in extremis (following blood sampling). Plasma concentrations of bivalirudin appeared to be linear and dose independent Clinical trial: Antithrombotic Effects of Ticagrelor Versus Clopidogrel .
Packing and Shipment :
Packing :plastic tube vials or lab box
Delivery details :within 3 days after payment .
Shipment :TNT EMS DHL etc
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